RESEARCH PROTOCOLS · PHARMACOKINETICS · HALF-LIFE

CJC-1295 Dosage in the Research Literature

Published dosing for CJC-1295 with DAC ranged from 30–125 μg/kg SC in human ascending-dose trials; the no-DAC form (Mod GRF 1-29), with its ~30-minute half-life, requires multiple daily pulses in research protocols. This page covers what was administered, to which species, at which dose, in published studies.

Important

All dosing described on this page is research-context framing: what was administered in a published study. This site makes no human dosing recommendations. CJC-1295 is not FDA-approved for any indication.

Research Doses Documented in the Literature

CJC-1295 dosage in published human trials was administered subcutaneously at 30, 60, 90, and 125 μg/kg in the ascending-dose Teichman 2006 trial[1] and at 60 and 90 μg/kg in the Ionescu and Frohman 2006 GH-pulsatility sub-study.[2] These are the only peer-reviewed human pharmacokinetic datasets for CJC-1295 with DAC. No published human pharmacokinetic study for CJC-1295 without DAC (Mod GRF 1-29) exists.

In the rodent literature, GHRH-knockout mice received 2 μg/day SC once-daily for five weeks in the Alba 2006 normalization study.[4] Ipamorelin, studied alongside CJC-1295 in stacking rationale research, was administered at 0.5–100 μg/kg SC, two to three times daily in rat bone and catabolism models.[10][11]

Half-Life: With DAC vs Without DAC

Ink-drawn plate of two abstract pharmacokinetic decay curves on hairline-ruled axes
PLATE II · PHARMACOKINETIC PROFILES: WITH-DAC vs NO-DAC

CJC-1295 without DAC has a plasma half-life of approximately 30 minutes, derived from DPP-IV resistance structural data; the Jetté et al. 2005 pharmacological characterization study is the primary source for this estimate.[3] CJC-1295 with DAC has a terminal half-life of 5.8–8.1 days, measured directly in the Teichman 2006 human ascending-dose trial.[1]

The difference — approximately 250-fold — arises entirely from the DAC albumin-binding mechanism: after SC injection, the maleimide-Lys C-terminal extension covalently binds the free thiol of Cys34 on circulating serum albumin, producing a ~70 kDa complex that is too large for renal filtration and is shielded from proteolytic inactivation. Sermorelin, for comparison, has a half-life of ~10–12 minutes.[15]

CJC-1295 with DAC vs Without DAC: Dosing Differences

The with-DAC and without-DAC forms require fundamentally different dosing architectures in research protocols.

Dosing Architecture Comparison
Form Half-Life Injection Frequency (Research) GH Profile
CJC-1295 with DAC 5.8–8.1 days Once-weekly or biweekly Tonic GH elevation
CJC-1295 no DAC (Mod GRF 1-29) ~30 minutes Multiple daily injections Discrete pulsatile GH response

With DAC — half-life 5.8–8.1 days — the Teichman 2006 study used single-injection ascending-dose designs; subsequent research has studied once-weekly or biweekly injection schedules.[1] The sustained tonic GH elevation means a single injection covers a week of measurable hormonal response.

Without DAC (Mod GRF 1-29) — half-life ~30 minutes — has a short activity window requiring multiple daily pulses in research designs to maintain sustained GH stimulation. Each injection produces a discrete GH pulse that decays within approximately one to two hours. This preserves physiologic pulsatility more closely than the tonic with-DAC profile.[14]

Injection Frequency in Research Protocols

CJC-1295 with DAC was studied at single-injection and once-weekly or biweekly SC injection schedules due to its 6–8 day half-life.[1][2] The no-DAC form (Mod GRF 1-29), with its ~30-minute half-life, requires multiple daily pulses in research protocols that aim to maintain continuous GH stimulation. Published clinical studies run from single-dose PK characterization to 49-day continuous administration.

Cycling Protocols in the Literature

Published research protocols vary in cycle design. Some studies use continuous administration for 12 or more weeks; others use 5-days-on / 2-days-off dosing schedules to model physiologic pulsatility rhythms. The Teichman 2006 trial ran to 49 days; the GHRH-knockout mouse study by Alba et al. used five continuous weeks of once-daily dosing.[1][4]

No peer-reviewed comparative cycling data exists in humans. The cycling question — how long to dose, how long to rest, whether rest is necessary — remains open in the published record.

Fasted vs Fed Administration

Insulin and elevated blood glucose blunt pituitary GH response via somatostatin-mediated suppression. Administering GHRH analogs in a fasted state is a recurring design variable in research studies examining GH pulse amplitude. This rationale is documented in both the Jetté 2005 and Teichman 2006 CJC-1295 papers.[1][3]

Reconstitution and Storage

Reconstituted peptide stability data for CJC-1295 in bacteriostatic water is not directly published for this compound. General peptide stability literature supports refrigerated storage at 2–8°C for up to approximately 30 days post-reconstitution for peptides of comparable size and structure. Freeze-thaw cycles risk peptide bond degradation and are generally avoided in laboratory protocols.